Angiology in Practice - download pdf or read online

By Susan B. Sherriff (auth.), Abdul-Majeed Salmasi, Antonio Strano (eds.)

ISBN-10: 9401062749

ISBN-13: 9789401062749

ISBN-10: 9401154066

ISBN-13: 9789401154062

Arterial and venous illnesses are significant factors of morbidity and mortality in lots of the global, particularly within the western hemisphere. not just of curiosity to angiologists, those health problems also are of shock to such a lot physicians in quite a few fields starting from cardiology, basic drugs and cardiovascular surgical procedure to body structure, pathology and scientific pharmacology. experts in diabetes, high blood pressure and epidemiology locate those health problems as not easy of their personal fields of curiosity because of the gross interrelation of those illnesses with their specialities.
This publication of 35 chapters includes an updated dialogue of varied arterial and venous health problems offering significant scientific functions starting from easy pathology, haemodynamics and haemorheology to medical good points and administration. distinctive cognizance has additionally been paid to epidemiology and prevention, discussing the entire matters involved. a unique part on vascular emergency has additionally been incorporated, thereby extending its usefulness to physicians and surgeons operating in coincidence and emergency units.

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Pathology of atherosclerosis 29 15. Parums DV, Brown DL, Mitchinson MJ. Serum antibodies to oxidized low-density lipoprotein and ceroid in chronic periaortitis. Arch Pathol Lab Med 1990; 114:383-7. 16. Faggiotto A, Ross R, Harker L. Studies of hypercholesteraemia in the nonhuman primate I. Changes that lead to fatty streak formation. Arteriosclerosis 1984;4:323-40. 17. Faggiotto A, Ross R. Studies of hypercholesteraemia in the nonhuman primate. II. Fatty streak conversion to fibrous plaque. Arteriosclerosis 1984;4:341-56.

This involves mUltiple mechanisms, apart from the likelihood of increased fibrin deposition, notably increased viscosity and enhanced platelet reactivity. Fibrinogen can be considered as an acute-phase inflammatory protein which is increased by numerous stimuli. These include smoking, diabetes, obesity and virtually any chronic inflammatory or infective state. Serum cholesterol and triglyceride concentration are also correlated with fibrinogen levels. 1 clearly illustrates, factor VII has a pivotal position in the coagulation cascade.

Although somewhat less complex than coagulation it is proving to have far more components than was originally thought. The ultimate product of the pathway is the proteolytic enzyme plasmin, which degrades fibrin in thrombi. The enzyme is derived from an inactive precursor, plasminogen, which is bound to fibrin and actually forms an integral part of the thrombus. Tissue plasminogen activator (t-PA), itself a protease synthesized and stored by endothelial cells, converts plasminogen to plasmin. It has a high affinity for fibrin, thereby helping to ensure that fibrinolysis is appropriately localized.

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Angiology in Practice by Susan B. Sherriff (auth.), Abdul-Majeed Salmasi, Antonio Strano (eds.)

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